Author

Katherine S. Ralston

University of California, DavisORCID 0000-0002-1604-7634Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Organism:E. coli· 1 Approach:genetic perturbation / CRISPR· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Rene L. SuleimaniD· 2 shared Wesley HuangiD· 2 shared Felina P. Loya· 1 shared Mary C. Irani· 1 shared Maura C. RuyechaniD· 1 shared Sylvia JebiwottiD· 1 shared Thomas J Luder· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

7 top collaborators · 10 mutual co-author links

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2 shared papersMicrobiology

Rene L. SuleimanORCID

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3 papers

Recent·Most cited

MicrobiologybioRxivPreprintJun 9, 2026

CRISPR/Cas9 knockout and editing of the major cysteine protease in Entamoeba histolytica

Wesley Huang, Rene L. Suleiman, Thomas J Luder…·Katherine S. Ralston

This study develops CRISPR/Cas9 for endogenous genome editing in Entamoeba histolytica, targeting the virulence factor CP5 and advancing genetic tools for the parasite.

DOI 10.64898/2026.06.08.731005

E. coliexperimental assay

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MicrobiologybioRxivPreprintMar 17, 2026

Human cell F-actin density differentially influences trogocytosis and phagocytosis by Entamoeba histolytica

Felina P. Loya, Mary C. Irani, Rene L. Suleiman, Katherine S. Ralston

This study investigates how human cell F-actin organization affects trogocytosis and phagocytosis by E. histolytica, finding differential impacts on these processes.

1 citation

humangenetic perturbation / CRISPR

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MicrobiologyInfection and ImmunityPeer-reviewedJul 31, 2025

Cross-species protection suggests Entamoeba histolytica trogocytosis enables complement resistance through the transfer of negative regulators of complement activation

Maura C. Ruyechan, Wesley Huang, Katherine S. Ralston

The paper shows that Entamoeba histolytica uses trogocytosis to acquire and display human complement regulators, enabling resistance to complement lysis even when exposed to mouse serum.

DOI 10.1128/iai.00220-25

humanexperimental assay

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