Author

Rachael M. Vollmer

University of Alabama at BirminghamORCID 0000-0002-1794-2278Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Rachael M. Vollmer1 paper · The University of Texas Southwestern Medical Center

Biology Disciplines

Click to filter author papers

Neuroscience· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Assay:electronic health records· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

Erik D. RobersoniD· 1 shared J. Nicholas CochraniD· 1 shared Jacob S Mesina· 1 shared Jeremy J. DayiD· 1 shared Jonathan R. RothiD· 1 shared Lori L. McMahoniD· 1 shared Mohammad WaqasiD· 1 shared Nancy V. N. CarulloiD· 1 shared

Activity by Year

2020-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperNeuroscience

Erik D. RobersonORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

NeuroscienceeLifePeer-reviewedJul 13, 2020

Alzheimer's disease risk gene BIN1 induces Tau-dependent network hyperexcitability

Yuliya Voskobiynyk, Jonathan R. Roth, J. Nicholas Cochran…·Erik D. Roberson

This study shows that increased BIN1 expression in neurons leads to hyperexcitability, which is modulated by Tau, suggesting a role for BIN1 in Alzheimer's disease pathogenesis.

70 citationstop 4%

humanelectronic health records

𝕏 PostBluesky