Author

Jonathan R. Roth

University of Alabama at BirminghamORCID 0000-0001-8978-4507Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Assay:electronic health records· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Erik D. RobersoniD· 2 shared J. Nicholas CochraniD· 2 shared Travis RushiD· 2 shared Adam R. Aldaher· 1 shared Jacob S Mesina· 1 shared Jeremy J. DayiD· 1 shared Lori L. McMahoniD· 1 shared Mohammad WaqasiD· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersNeuroscience

Erik D. RobersonORCID

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2 papers

Recent·Most cited

NeuroscienceeLifePeer-reviewedJul 13, 2020

Alzheimer's disease risk gene BIN1 induces Tau-dependent network hyperexcitability

Yuliya Voskobiynyk, Jonathan R. Roth, J. Nicholas Cochran…·Erik D. Roberson

This study shows that increased BIN1 expression in neurons leads to hyperexcitability, which is modulated by Tau, suggesting a role for BIN1 in Alzheimer's disease pathogenesis.

70 citationstop 4%

humanelectronic health records

𝕏 PostBluesky

NeuroscienceNeurobiology of DiseasePeer-reviewedNov 4, 2019

A peptide inhibitor of Tau-SH3 interactions ameliorates amyloid-β toxicity

Travis Rush, Jonathan R. Roth, Samantha J. Thompson…·Erik D. Roberson

The paper develops a peptide inhibitor of Tau-SH3 interactions and shows it reduces Aβ oligomer toxicity in neurons.

30 citations

ratproteomics

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