Author

Mohammad Waqas

Massachusetts General HospitalORCID 0000-0002-4633-4167Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetic & Genomic Medicine· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Assay:electronic health records· 1 Approach:experimental assay· 1 Research Focus:GWAS / association mapping· 1 Approach:statistical model· 1

Frequent Co-Authors

Dmitry ProkopenkoiD· 1 shared Erik D. RobersoniD· 1 shared J. Nicholas CochraniD· 1 shared Jacob S Mesina· 1 shared Jeremy J. DayiD· 1 shared Jonathan R. RothiD· 1 shared Julian WillettiD· 1 shared Kristina Mullin· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperGenetic & Genomic Medicine

Dmitry ProkopenkoORCID

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2 papers

Recent·Most cited

Identification of 16 novel Alzheimer's disease susceptibility loci using multi-ancestry meta-analyses of clinical Alzheimer's disease and AD-by-proxy cases from four whole genome sequencing datasets

Julian Willett, Mohammad Waqas, Younjung Choi, Tiffany Ngai, Kristina Mullin, Rudolph E. Tanzi, Dmitry Prokopenko

This study identifies 16 new Alzheimer's disease susceptibility loci through multi-ancestry GWAS using whole genome sequencing data from diverse cohorts.

medRxiv · Sep 11, 2024 · PreprintGenetic & Genomic Medicine

Alzheimer's disease risk gene BIN1 induces Tau-dependent network hyperexcitability

Yuliya Voskobiynyk, Jonathan R. Roth, J. Nicholas Cochran, Travis Rush, Nancy V. N. Carullo, Jacob S Mesina, Mohammad Waqas, Rachael M. Vollmer, Jeremy J. Day, Lori L. McMahon, Erik D. Roberson

This study shows that increased BIN1 expression in neurons leads to hyperexcitability, which is modulated by Tau, suggesting a role for BIN1 in Alzheimer's disease pathogenesis.

eLife · Jul 13, 2020Peer-reviewedNeuroscience