Jana Fiona Liewald

This study identifies NRA-1 as an activity-dependent regulator of cholinergic signalling and sensory integration in C. elegans, showing it modulates receptor gating and behaviour.

The paper uses all-optical methods to study gap junction coupling in C. elegans muscle cells, showing how specific innexins affect synchronization and locomotion.

This study investigates the role of FLWR-1 in synaptic vesicle recycling and neuronal activity in C. elegans, finding it regulates Ca2+ levels via PMCA and affects excitation-inhibition balance.

The paper shows that loss of neuropeptide signaling in C. elegans leads to increased muscle excitability via CaV1 channels to compensate for reduced cholinergic transmission.

The paper investigates neuropeptide regulation of neuromuscular signaling in zebrafish, showing that cAMP signaling enhances synaptic transmission and that neuropeptide mutants exhibit altered locomotion and synaptic properties.

The paper investigates how RIM and RIM-binding proteins regulate synaptic transmission by localizing Cav2 channels in C. elegans neuromuscular junctions, showing differential effects on cholinergic and GABAergic transmission.

This paper introduces an optogenetic voltage-clamp technique for precise control of cellular voltage in live animals, demonstrating its application in C. elegans and rat neurons.

This paper introduces optoSynC, an optogenetic tool that rapidly and reversibly silences synaptic transmission by clustering synaptic vesicles in C. elegans.

This study shows that synapsin SNN-1 is required for cAMP-dependent neuropeptide release in C. elegans motor neurons by capturing dense core vesicles at synapses.

The paper evaluates voltage imaging tools in C. elegans, showing voltage sensitivity and depolarization differences in muscles and pharynx.

This study identifies the functional role of AS motor neurons in coordinating locomotion in C. elegans by integrating signals from premotor interneurons and mediating body wave propagation.