Author

Jonas Sørensen

University of CopenhagenORCID 0000-0002-2055-0953Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:proteomics· 3 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Andrea IrazokiiD· 3 shared Cathrine Knos· 3 shared Christian T. VoldstedlundiD· 3 shared Edmund BatteyiD· 3 shared Johanne Louise ModvigiD· 3 shared Lykke SylowiD· 3 shared Steffen Henning RauniD· 3 shared Christian Strini CarliD· 2 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersMolecular Biology

Andrea IrazokiORCID

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3 papers

Recent·Most cited

Systems BiologybioRxivPreprintJun 15, 2026

Plasma proteomics identifies an IL-6 -associated SAA axis linked to muscle wasting and survival in patients with cancer cachexia

Jonas Sørensen, Christian T. Voldstedlund, Zakarias Kefil Jensen Ogueboule…·Lykke Sylow

The paper identifies an IL-6-associated SAA axis linked to muscle wasting and poor survival in cancer cachexia through plasma proteomics and functional studies.

DOI 10.64898/2026.06.11.730788

humanproteomics

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Molecular BiologybioRxivPreprintNov 26, 2025

Proteomic and phospho-proteomic longitudinal signatures of human skeletal muscle in lung cancer cachexia

Jonas Sørensen, Christian T. Voldstedlund, Edmund Battey…·Lykke Sylow

The paper identifies proteomic and phosphoproteomic changes in skeletal muscle of lung cancer patients with cachexia, linking them to muscle wasting and signaling pathways.

DOI 10.1101/2025.11.26.690647

humanproteomics

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Molecular BiologybioRxivPreprintNov 19, 2025

Pathophysiological remodeling of the skeletal muscle microenvironment in patients with lung cancer

Edmund Battey, Andrea Irazoki, Jakob Wang…·Lykke Sylow

The paper investigates muscle remodeling in NSCLC patients, identifying changes in fibro-adipogenic progenitors, mitochondrial function, and immune cells that contribute to muscle wasting.

1 citation

humanproteomics

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