Jakob Wang

The paper identifies CD90 as a marker distinguishing two muscle stem cell subpopulations with different activation kinetics and quiescence maintenance mechanisms in mice and humans.

This study examines how exercise-induced hypertrophy in older adults alters fibro-adipogenic progenitors (FAPs) and macrophages in skeletal muscle, revealing phenotypic shifts and interactions that support muscle growth.

This study uses a rat model to show that MuSK autoimmunity causes fiber-type-selective muscle atrophy and proteome remodeling, with mitochondrial and translational dysregulation as key features.

The paper investigates muscle remodeling in NSCLC patients, identifying changes in fibro-adipogenic progenitors, mitochondrial function, and immune cells that contribute to muscle wasting.

This study identifies fibro-adipogenic progenitors and macrophages as key players in elderly human skeletal muscle regeneration through spatial and single-cell transcriptomics, revealing their interplay via complement factor C3.