Author

Marjolein J. W. de Bruijn

Erasmus MCORCID 0000-0002-2142-4684Unclaimed ORCID profile · follow to get notified of new preprints

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Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Approach:machine learning (classical)· 1 Assay:scRNA-seq· 1

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Anne Onrust-van SchoonhoveniD· 1 shared Esmee K. van der PloegiD· 1 shared Geert H. van WeeldeniD· 1 shared Gregory van BeekiD· 1 shared James P. Di SantoiD· 1 shared Jochem Weekers· 1 shared Laura SuraceiD· 1 shared Lianne TrapiD· 1 shared

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2026-2026 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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1 shared paperImmunology

Anne Onrust-van SchoonhovenORCID

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1 paper

Recent·Most cited

Common γ-chain cytokines induce an epigenomically plastic precursor-like KIT+ ILC2 state linked to immune disease susceptibility

Simone E. M. Olsthoorn, Anne Onrust-van Schoonhoven, Marjolein J. W. de Bruijn, Jochem Weekers, Menno van Nimwegen, Gregory van Beek, Willem de Koning, Lianne Trap, Esmee K. van der Ploeg, Geert H. van Weelden, Mathijs A. Sanders, Laura Surace, James P. Di Santo, Rudi W. Hendriks, Ralph Stadhouders

This study identifies KIT+ ILC2s as an epigenomically plastic state linked to immune disease susceptibility and shows that STAT5 activation by cytokines induces this phenotype.

Journal of Allergy and Clinical Immunology · Jun 17, 2026Peer-reviewedImmunology