Author

Mathijs A. Sanders

Erasmus MC

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:human· 2 Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1

Frequent Co-Authors

Adil S. al Hinai· 1 shared Andrew W. Roberts· 1 shared Anne Onrust-van SchoonhoveniD· 1 shared Annelieke Zeilemaker· 1 shared Ashish Bajel· 1 shared Bob Löwenberg· 1 shared Bram Luiken· 1 shared Christoffer FlensburgiD· 1 shared

Activity by Year

2017-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperCancer Biology

Adil S. al Hinai

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Common γ-chain cytokines induce an epigenomically plastic precursor-like KIT+ ILC2 state linked to immune disease susceptibility

Simone E. M. Olsthoorn, Anne Onrust-van Schoonhoven, Marjolein J. W. de Bruijn, Jochem Weekers, Menno van Nimwegen, Gregory van Beek, Willem de Koning, Lianne Trap, Esmee K. van der Ploeg, Geert H. van Weelden, Mathijs A. Sanders, Laura Surace, James P. Di Santo, Rudi W. Hendriks, Ralph Stadhouders

This study identifies KIT+ ILC2s as an epigenomically plastic state linked to immune disease susceptibility and shows that STAT5 activation by cytokines induces this phenotype.

Journal of Allergy and Clinical Immunology · Jun 17, 2026Peer-reviewedImmunology

Germline loss of MBD4 predisposes to leukaemia due to a mutagenic cascade driven by 5mC

Mathijs A. Sanders, Edward Chew, Christoffer Flensburg, Annelieke Zeilemaker, Sarah E. Miller, Adil S. al Hinai, Ashish Bajel, Bram Luiken, Melissa Rijken, Tamara Mclennan, Remco M. Hoogenboezem, François G. Kavelaars, Marnie E. Blewitt, Eric M. Bindels, Warren S. Alexander, , Ian J. Majewski

This study shows that germline MBD4 loss causes AML with high C>T mutations due to 5mC deamination, and highlights interactions with somatic DNMT3A mutations.

bioRxiv · Nov 1, 2017 · PreprintCancer Biology