Author

Giuseppe Locatelli

Novartis (Switzerland)ORCID 0000-0002-2179-4407Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:mouse· 2 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Almir AljovićiD· 1 shared Arek KendirliiD· 1 shared Caroline FecheriD· 1 shared Daniel EngelsiD· 1 shared Delphine TheodorouiD· 1 shared Don MahadiD· 1 shared Doron MerkleriD· 1 shared Eliane Pierrel· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperNeuroscience

Almir AljovićORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Experimental Autoimmune Encephalomyelitis Causes Skeletal Muscle Dysfunction in Mice

Julian Boesch, Pamela Ramseier, Sarah Tisserand, Eliane Pierrel, Giuseppe Locatelli, Serge Summermatter

This study shows that Experimental Autoimmune Encephalomyelitis (EAE) in mice causes skeletal muscle dysfunction, as evidenced by reduced muscle force during tetanic contractions, highlighting similarities with human MS.

Frontiers in Neurology · Apr 8, 2025Peer-reviewedPhysiology

Targeting the TCA cycle can ameliorate widespread axonal energy deficiency in neuroinflammatory lesions

Yi-Heng Tai, Daniel Engels, Giuseppe Locatelli, Ioanna Emmanouilidis, Caroline Fecher, Delphine Theodorou, Stephan A. Müller, Simon Licht‐Mayer, Mario Kreutzfeldt, Ingrid Wagner, Natália Prudente de Mello, Sofia-Natsouko Gkotzamani, Laura Trovò, Arek Kendirli, Almir Aljović, , Thomas Misgeld

The paper shows that TCA cycle dysfunction contributes to axonal energy deficiency in neuroinflammatory lesions and that restoring TCA enzymes can alleviate this deficit.

Nature Metabolism · Jul 10, 2023Peer-reviewedNeuroscience