Author

Xiangyu Zhao

ShanghaiTech UniversityORCID 0000-0002-2186-476XUnclaimed ORCID profile · follow to get notified of new preprints

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Xiangyu Zhao2 papers · ShanghaiTech University

Biology Disciplines

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Bioinformatics· 2 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:deep learning· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1 Research Focus:variant calling· 1 Assay:whole-exome sequencing· 1

Frequent Co-Authors

Xuesong LiuiD· 3 shared Chenxu WuiD· 2 shared Kaixuan DiaoiD· 2 shared Shixiang WangiD· 2 shared Tao WuiD· 2 shared Xiaoqin SuniD· 2 shared Casimiro GerarduzziiD· 1 shared Dongsheng Bai· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 34 mutual co-author links

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3 shared papersBioinformatics

Xuesong LiuORCID

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3 papers

Recent·Most cited

The repertoire of copy number alteration signatures in human cancer

Ziyu Tao, Shixiang Wang, Chenxu Wu, Tao Wu, Xiangyu Zhao, Ning Wei, Guangshuai Wang, Jinyu Wang, Jing Chen, Kaixuan Diao, Fuxiang Chen, Xuesong Liu

This study identifies 14 CNA signatures from pan-cancer WGS and TCGA SNP array data, revealing novel patterns and prognostic implications for cancer patients.

Briefings in Bioinformatics · Jan 28, 2023Peer-reviewedBioinformatics

Seq2Neo: a comprehensive pipeline for cancer neoantigen im-munogenicity prediction

Kaixuan Diao, Jing Chen, Tao Wu, Xuan Wang, Guangshuai Wang, Xiaoqin Sun, Xiangyu Zhao, Chenxu Wu, Jinyu Wang, Huizi Yao, Casimiro Gerarduzzi, Xuesong Liu

Seq2Neo predicts neoantigen immunogenicity using a CNN model trained on genomic alterations, outperforming existing tools in independent datasets.

International Journal of Molecular Sciences · Oct 1, 2022Peer-reviewedBioinformatics

Essential role for Ggct in erythrocyte antioxidant defense

Zaoke He, Xiaoqin Sun, Shixiang Wang, Dongsheng Bai, Xiangyu Zhao, Ying Han, Piliang Hao, Xuesong Liu

This study shows that Ggct deletion in mice causes splenomegaly and anemia due to oxidative damage and reduced glutathione in red blood cells.

bioRxiv · Jun 21, 2021 · PreprintGenetics