Author

Stefania D’Adamo

University of BolognaORCID 0000-0002-2209-9158Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

Cristina CosentinoiD· 2 shared Francesco AlabisoiD· 2 shared Romano RegazziiD· 2 shared Silvia CetrulloiD· 2 shared Véronique Menoud· 2 shared A. GalliiD· 1 shared Aleksandra Seragnoli ChystyakovaiD· 1 shared Andrea GiaccariiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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2 shared papersCell Biology

Cristina CosentinoORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

5'tRNA-derived fragments modulate β-cell homeostasis and islet macrophage activation in type 2 diabetes

Cristina Cosentino, Rémy Klein, Véronique Menoud, Claudiane Guay, Elena Aiello, Stefano Auddino, Gianfranco Di Giuseppe, Gea Ciccarelli, A. Galli, Francesco Alabiso, Eleonora Mangano, Flora Brozzi, Karim Bouzakri, Stefania D’Adamo, Silvia Cetrullo, , Romano Regazzi

This study identifies 5'tRNA-derived fragments as mediators of β-cell dysfunction and islet macrophage activation in type 2 diabetes.

Nature Communications · May 4, 2026Peer-reviewedCell Biology

3'tRNAAsp(GTC)-derived fragment links inflammation to post-transcriptional reprogramming in chondrocytes during osteoarthritis.

Francesco Alabiso, Aleksandra Seragnoli Chystyakova, Cristina Cosentino, Irene Bissoli, Véronique Menoud, Ilaria Bedei, Merissa Olmer, Veronica Panichi, Isabella Rusciano, Paolo Dolzani, Carla Renata Arciola, Stefano Ratti, Martin Lotz, Rosa Maria Borzı̀, Romano Regazzi, , Stefania D’Adamo

This study identifies 3'tRNAAsp(GTC)-derived fragment as a regulator linking inflammation to translational reprogramming in chondrocytes during osteoarthritis.

bioRxiv · Jan 23, 2026 · PreprintBiochemistry