Author

Michelle Mosqueda

University of Massachusetts Chan Medical SchoolORCID 0000-0002-2284-0399Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

Cathleen M. LutziD· 1 shared Daryl A. BoscoiD· 1 shared Debra Cameron· 1 shared Del Hayden GaddiD· 1 shared Dorothy P. SchaferiD· 1 shared Elaine T. LimiD· 1 shared Francesca MassiiD· 1 shared Jeffrey A. NickersoniD· 1 shared

Activity by Year

2024-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Cathleen M. LutzORCID

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1 paper

Recent·Most cited

Expression of ALS-PFN1 impairs vesicular degradation in iPSC-derived microglia

Salome Funes, Jonathan Jung, Del Hayden Gadd, Michelle Mosqueda, Jianjun Zhong, Shankaracharya, Matthew Unger, Karly M. Stallworth, Debra Cameron, Melissa S. Rotunno, Pepper Dawes, Megan Fowler, Pamela J. Keagle, Justin A. McDonough, Sivakumar Boopathy, , Daryl A. Bosco

This study shows that ALS-linked PFN1 mutations impair vesicular degradation in iPSC-derived microglia by disrupting autophagy, which can be mitigated by Rapamycin.

Nature Communications · Mar 20, 2024Peer-reviewedNeuroscience