Author

Tal Levin

Weizmann Institute of ScienceORCID 0000-0002-2301-3168Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:bacteria (other)· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Assay:protein structure· 1 Research Focus:protein structure prediction· 1 Assay:proteomics· 1

Frequent Co-Authors

Emmanuel D. LevyiD· 2 shared Alexander I. AlexandroviD· 1 shared Arseniy A. LoboviD· 1 shared Benjamin DubreuiliD· 1 shared Bruno E. CorreiaiD· 1 shared Casper A. GoverdeiD· 1 shared Derek N. WoolfsoniD· 1 shared Dikla Levi· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

Emmanuel D. LevyORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Profiling the physiological impact of aberrant folded-state protein filamentation in cells

Tal Levin, Hector Garcia‐Seisdedos, Arseniy A. Lobov, Matthias Wojtynek, Alexander I. Alexandrov, Ghil Jona, Dikla Levi, Ohad Medalia, Emmanuel D. Levy

This study investigates the physiological impact of folded-state protein agglomerates in yeast, finding they do not disrupt proteostasis or cell fitness and show minimal effects on cellular processes.

Molecular Systems Biology · Sep 15, 2025Peer-reviewedCell Biology

An atlas of protein homo-oligomerization across domains of life

Hugo Schweke, Martin Pačesa, Tal Levin, Casper A. Goverde, Prasun Kumar, Yoan Duhoo, L Dornfeld, Benjamin Dubreuil, Sandrine Georgeon, Sergey Ovchinnikov, Derek N. Woolfson, Bruno E. Correia, Sucharita Dey, Emmanuel D. Levy

The paper predicts homo-oligomerization across four proteomes using AlphaFold2, finding that 50% of archaea, 45% of bacteria, and 20% of eukaryotes form homomers, with disease mutations enriched at interfaces.

Cell · Feb 1, 2024Peer-reviewedBioinformatics