Fei Ran Li

The paper examines structural asymmetry abnormalities in young children with autism and developmental delay/intellectual disability using normative models and finds group-specific patterns linked to gene expression and behavioral outcomes.

The paper shows that high emotional reactivity in mice is linked to a distinct hippocampal-amygdala circuit influenced by glucocorticoid receptor overexpression.

The paper shows that inhibiting VTA dopamine neurons during reward cue presentation prevents sign-tracking behavior, confirming dopamine's role in encoding incentive value.

This paper investigates the causal effect of puberty timing on plasma metabolites using Mendelian randomization and finds moderate evidence of causal relationships for 16 metabolites after adjusting for confounders.

This study identifies a SENP1 mutation in mice that causes core autistic-like symptoms by disrupting the SENP1-FMRP axis in the retrosplenial agranular cortex.