Mark Basik

This study identifies TACC3 as a coordinator of translation and epitranscriptomic reprogramming that determines cancer cell sensitivity or tolerance to mitotic stress induced by microtubule targeting agents.

This study identifies ATR and PKMYT1 as targets to overcome carboplatin resistance in TNBC by re-sensitizing resistant models to carboplatin through mitotic catastrophe.

ProCanFDL uses federated deep learning to subtype cancers using proteomic data while preserving privacy, achieving performance gains over local models.