Author

Larry W. Markham

Indiana University HealthORCID 0000-0002-2804-4641Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Assay:imaging· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Alyssa M. Richards· 2 shared Areli Jannes S. JavieriD· 2 shared Conner C. EarliD· 2 shared Craig J. GoergeniD· 2 shared Steven S. WelciD· 2 shared Akanksha AgrawaliD· 1 shared B. Zeidler· 1 shared Carol A. WitczakiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersPhysiology

Alyssa M. Richards

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Empagliflozin preserves cardiac function and modulates metabolism in a mouse model of Duchenne muscular dystrophy

B. Zeidler, Connor R Thomas, John P. Salvas, Areli Jannes S. Javier, Alyssa M. Richards, Linda A. Bean, Conner C. Earl, Akanksha Agrawal, Niharika Narra, Li‐Fan Zeng, Carol A. Witczak, Joshua R. Huot, Il‐man Kim, Meena S. Madhur, Mark C. Kowala, , Steven S. Welc

This study shows empagliflozin improves cardiac function and mitochondrial health in a mouse model of Duchenne muscular dystrophy.

bioRxiv · Mar 14, 2026 · PreprintPhysiology

Functional cardiac consequences of β-adrenergic stress-induced injury in the mdx mouse model of Duchenne muscular dystrophy

Conner C. Earl, Areli J. Javier, Alyssa M. Richards, Larry W. Markham, Craig J. Goergen, Steven S. Welc

This paper introduces a method using low-dose isoproterenol to enhance cardiac dysfunction in mdx mice, showing increased myocyte injury, fibrosis, and reduced ventricular function compared to wild-type mice.

bioRxiv · Apr 15, 2024 · PreprintPhysiology