Author

Irina Nekrasova

Institut de Biologie de l'École Normale SupérieureORCID 0000-0002-3121-9772Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Genomics· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:statistical model· 2 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:genome assembly· 1 Organism:human· 1 Assay:proteomics· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

Alexey A. PotekhiniD· 1 shared Amandine TouzeauiD· 1 shared Andrea FrapportiiD· 1 shared Andreea NissenkorniD· 1 shared Arnaud CoulouxiD· 1 shared Augustin de Vanssay· 1 shared Caridad Miró-PinaiD· 1 shared Coralie ZangarelliiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperGenomics

Alexey A. PotekhinORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

The tiny germline chromosomes of Paramecium aurelia have anexceptionally high recombination rate and are capped by a new class of Helitrons

Olivier Arnaiz, Frédéric Guèrin, Arnaud Couloux, Caridad Miró-Pina, Guillaume Pellerin, Irina Nekrasova, Joëlle Amselem, Jean‐Marc Aury, Simran Bhullar, Andrea Frapporti, Emmanuelle Lerat, Isabelle Luyten, Sophie Malinsky, Nathalie Mathy, Alexey A. Potekhin, , Linda Sperling

The paper reports the genome of Paramecium aurelia, revealing tiny germline chromosomes with an exceptionally high recombination rate and a new class of Helitrons at their ends.

BMC Biology · Apr 16, 2026Peer-reviewedGenomics

Deciphering the mechanistic basis for the pathological effect of the Gαo E246K mutation in neurodevelopmental disorder

Isra Sadiya, Irina Nekrasova, Meirav Avital‐Shacham, Naomi van Wijk, Keren Zohar, Nir Kalisman, Dina Schneidman‐Duhovny, Ehud Banne, Andreea Nissenkorn, Lubov Blumkin, Michal Linial B, Mickey Kosloff

The paper investigates the molecular mechanism of the Gαo E246K mutation in neurodevelopmental disorders, showing it disrupts the GTPase cycle by impairing Gβγ dissociation, leading to dominant-negative effects.

bioRxiv · Sep 3, 2025 · PreprintNeuroscience