Author

Jacopo Zasso

University of TrentoORCID 0000-0002-3151-6443Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetics· 1 Molecular Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Luciano ContiiD· 2 shared Alan MonzianiiD· 1 shared Alessandra Zappulo· 1 shared Alessandro CutarelliiD· 1 shared Ana KotteiD· 1 shared Blagoje SoskiciD· 1 shared Christoph DieterichiD· 1 shared Dilara AyyildiziD· 1 shared

Activity by Year

2018-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersGenetics

Luciano ContiORCID

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3 papers

Recent·Most cited

Insulin synthesis is sustained by Tent5 poly(A) polymerases

Ana Kotte, Fabio Marcuccio, Linda Masante, Nadja von Wiegen, Lucia Prandi, Raquel Sofia Silva, Rodrigo Pracana, Jacopo Zasso, Blagoje Soskic, Alessandra Zappulo, Ivano Legnini

The paper identifies Tent5 poly(A) polymerases as stabilizers of insulin mRNA, showing their specific activity regulated by glucose and localization.

bioRxiv · Jul 6, 2026 · PreprintMolecular Biology

Defective linear and circular RNAs biogenesis in Huntington's disease: CAG repeat expansion hijacks neuronal splicing

Dilara Ayyildiz, Alan Monziani, Takshashila Tripathi, Jessica Döring, Guendalina Bergonzoni, Emanuela Kerschbamer, Francesca Di Leva, Elia Pennati, Luisa Donini, Marina V. Kovalenko, Jacopo Zasso, Luciano Conti, Vanessa Chantal Wheeler, Christoph Dieterich, Silvano Piazza, , Marta Biagioli

The paper shows that CAG repeat expansion in Huntington's disease disrupts splicing, reducing circular RNA production and affecting neural progenitors and striatum before symptoms appear.

bioRxiv · Dec 27, 2021 · PreprintGenetics

Inducible alpha-synuclein overexpression affects human Neural Stem Cells behavior

Jacopo Zasso, Mastad Ahmed, Alessandro Cutarelli, Luciano Conti

This paper develops a conditional human neural stem cell model to study alpha-synuclein effects on cell behavior and toxicity in Parkinson's disease.

Stem Cells and Development · Apr 19, 2018Peer-reviewedNeuroscience