Author

Ali Mirzaesmaeili

Hospital for Sick ChildrenORCID 0000-0002-3169-5066Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Organism:rat· 1

Frequent Co-Authors

Ali A. Abdul SateriD· 1 shared Allen Volchuk· 1 shared B. MartinsiD· 1 shared Benjamin Ethan SteinbergiD· 1 shared Blayne Amir SayediD· 1 shared Claire J. Hur· 1 shared Danish AliiD· 1 shared Daria TaskinaiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperImmunology

Ali A. Abdul SaterORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

ImmunologybioRxivPreprintFeb 10, 2026

NINJ1 mediates hepatic ischemia-reperfusion injury

Jan Mossemann, B. Martins, Y. Zhao…·Blayne Amir Sayed

This study identifies NINJ1 as a mediator of hepatic ischemia-reperfusion injury, showing its role in cell death and inflammation, and suggests it as a therapeutic target.

DOI 10.64898/2026.02.07.704585

humanmouse

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ImmunologyJournal of AutoimmunityPeer-reviewedFeb 5, 2025

Selective disruption of Traf1/cIAP2 interaction attenuates inflammatory responses and limits sepsis and rheumatoid arthritis

Yitian Tang, Fatemah Aleithan, Sahib Singh Madahar…·Ali A. Abdul Sater

This study identifies a TRAF1 mutation that disrupts its interaction with cIAP2, reducing inflammation and protecting against sepsis and rheumatoid arthritis.

15 citationstop 4%

mouseexperimental assay

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