Author

Kavindra Nath

University of PennsylvaniaORCID 0000-0002-3197-5703Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Assay:metabolomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Assay:metagenomics· 1 Assay:proteomics· 1

Frequent Co-Authors

Aaron R. GoldmaniD· 2 shared Cosimo LobelloiD· 2 shared David RushmoreiD· 2 shared Johnvesly BasappaiD· 2 shared Mariusz A. WasikiD· 2 shared Reza NejatiiD· 2 shared Shengchun WangiD· 2 shared David S. Nelson· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

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2 shared papersCancer Biology

Aaron R. GoldmanORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintOct 4, 2026

A Novel ACLY Inhibitor Induces Transcriptional and Metabolic Reprogramming to Overcome Tyrosine kinase inhibitor Resistance in Lymphoma

Nagarajan Pattabiraman, Cosimo Lobello, David Rushmore…·Johnvesly Basappa

This paper identifies VCB1, a novel ACLY inhibitor, which overcomes TKI resistance in lymphoma by disrupting lipid and nucleotide metabolism, ISR, and redox homeostasis.

DOI 10.64898/2026.09.29.755331

humanproteomics

𝕏 PostBluesky

Cancer BiologyCancer LettersPeer-reviewedApr 7, 2026

Targeting the Mitochondrial ACSS1 Dependent Acetate and Pyrimidine Axis Suppresses Mantle Cell Lymphoma Progression

Johnvesly Basappa, Aaron R. Goldman, Cosimo Lobello…·Mariusz A. Wasik

This paper identifies ACSS1 as a metabolic driver in lymphomas, showing its role in acetate-to-acetyl-CoA conversion and pyrimidine biosynthesis, and demonstrates that targeting ACSS1 suppresses tumor growth.

2 citations

humanmetabolomics

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