Author

Anuja R. Bony

University of WollongongORCID 0000-0002-3214-8814Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:electronic health records· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1

Frequent Co-Authors

David J. AdamsiD· 2 shared Jeffrey R. McArthuriD· 1 shared Mariana BrizuelaiD· 1 shared Rocio K. Finol‐UrdanetaiD· 1 shared Sonia Garcia‐CaraballoiD· 1 shared Stuart M. BrierleyiD· 1 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

6 top collaborators · 9 mutual co-author links

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2 shared papersNeuroscience

David J. AdamsORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintMar 21, 2025

Differential contributions of CaV2.2, GIRK, and HCN channels to the modulation of excitability by alpha-conotoxin Vc1.1 and baclofen effects in somatic and visceral sensory neurons

Mariana Brizuela, Anuja R. Bony, Sonia Garcia‐Caraballo…·Stuart M. Brierley

This study investigates how CaV2.2, GIRK, and HCN channels modulate excitability in somatic and visceral sensory neurons in response to Vc1.1 and baclofen, finding distinct mechanisms in different neuron types.

DOI 10.1101/2025.03.21.644483

mousehuman

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NeuroscienceBritish Journal of PharmacologyPeer-reviewedOct 2, 2021

Analgesic α-conotoxins modulate GIRK1/2 channels via GABAB receptor activation and reduce neuroexcitability

Anuja R. Bony, Jeffrey R. McArthur, Rocio K. Finol‐Urdaneta, David J. Adams

This study shows that Vc1.1 modulates GIRK1/2 channels via GABAB receptor activation, reducing neuronal excitability and providing insights for non-opioid pain treatments.

17 citations

mouseelectronic health records

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