Author

Yifei Cai

Columbia UniversityORCID 0000-0003-0108-8166Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:ChIP-seq· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1

Frequent Co-Authors

Amber Braker· 1 shared Anastasia DiamantopoulouiD· 1 shared Angus Clark NairniD· 1 shared Anita J HuttneriD· 1 shared Atsushi TakataiD· 1 shared Bin XuiD· 1 shared Chia‐Yuan ChangiD· 1 shared Enrico Cannavò· 1 shared

Activity by Year

2019-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperNeuroscience

Amber Braker

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Subcellular proteomics and iPSC modeling uncover reversible mechanisms of axonal pathology in Alzheimer's disease

Yifei Cai, Jean E. Kanyo, Rashaun S. Wilson, Shveta Bathla, Pablo Leal Cardozo, Лей Тонг, Shanshan Qin, Lukas A. Fuentes, Iguaracy Pinheiro‐de‐Sousa, Tram Huynh, Liyuan Sun, Mohammad Shahid Mansuri, Zichen Tian, Hao-Ran Gan, Amber Braker, , Jaime Grutzendler

The paper uses subcellular proteomics and iPSC models to identify mTOR pathway activation in axonal spheroids and show its reversible role in Alzheimer's disease pathology.

Nature Aging · Mar 10, 2025Peer-reviewedNeuroscience

Recapitulation and reversal of schizophrenia-related phenotypes in Setd1a-deficient mice

Jun Mukai, Enrico Cannavò, Gregg W. Crabtree, Ziyi Sun, Anastasia Diamantopoulou, Pratibha Thakur, Chia‐Yuan Chang, Yifei Cai, Stavros Lomvardas, Atsushi Takata, Bin Xu, Joseph A. Gogos

This study shows that Setd1a-deficient mice exhibit schizophrenia-like phenotypes, which can be reversed by restoring Setd1a expression or inhibiting LSD1.

Neuron · Oct 9, 2019Peer-reviewedNeuroscience