Transient protein phosphorylation promotes disease tolerance to sepsis
Wolfgang Vivas, Johannes Roth, Katharina Willman, Hjalmar R. Bouma, Franziska Röstel, Karen Dlubatz, Gianna Hirth, Nadine Pömpner, Therese Dau, Emilio Cirri, Sheng Zhang, Mirko Peitzsch, Triantafyllos Chavakis, Michael W Bauer, Gianni Pannagiotou, , Sebastian Weis
This paper identifies transient GSK3β activation as a mechanism promoting disease tolerance to sepsis through hypometabolism, without affecting pathogen burden.
bioRxiv · Jun 10, 2026 · PreprintImmunology