Author

Eirini Antoniou

Karolinska InstitutetORCID 0000-0002-3616-9049Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Assay:ChIP-seq· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

Johan HolmbergiD· 2 shared Behzad Yaghmaeian SalmaniiD· 1 shared Erik SödersteniD· 1 shared Henrik LindehelliD· 1 shared Ilknur Safak Demirel· 1 shared Iria Laura MartineziD· 1 shared Jakob J. E. StenmaniD· 1 shared Jiacheng ZhuiD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCancer Biology

Johan HolmbergORCID

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2 papers

Recent·Most cited

Cancer BiologyProceedings of the National Academy of SciencesPeer-reviewedOct 21, 2025

HIF2α negatively regulates MYCN protein levels and promotes a low-risk noradrenergic phenotype in neuroblastoma.

Juan Yuan, Subhamita Maitra, Eirini Antoniou…·Johan Holmberg

This paper shows that HIF2α reduces MYCN protein levels and promotes a low-risk noradrenergic phenotype in neuroblastoma, suggesting HIF2 has tumor suppressor rather than oncogenic function.

1 citation

humanbulk RNA-seq

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NeuroscienceScience AdvancesPeer-reviewedAug 26, 2022

PRC2-mediated repression is essential to maintain identity and function of differentiated dopaminergic and serotonergic neurons

Konstantinos Toskas, Behzad Yaghmaeian Salmani, Olga Skiteva…·Johan Holmberg

This study shows that PRC2-mediated repression is essential for maintaining identity and function of dopaminergic and serotonergic neurons, with loss leading to behavioral deficits.

24 citations

mouseChIP-seq

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