Author

Erik Södersten

Karolinska InstitutetORCID 0000-0002-2998-4889Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:mouse· 2 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

Johan HolmbergiD· 2 shared Behzad Yaghmaeian SalmaniiD· 1 shared David YudovichiD· 1 shared Diahann A. M. AtachoiD· 1 shared Eirini AntoniouiD· 1 shared Jenny G Johansson· 1 shared Johan JakobssoniD· 1 shared Jonas LarssoniD· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersNeuroscience

Johan HolmbergORCID

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2 papers

Recent·Most cited

PRC2-mediated repression is essential to maintain identity and function of differentiated dopaminergic and serotonergic neurons

Konstantinos Toskas, Behzad Yaghmaeian Salmani, Olga Skiteva, Wojciech Pasławski, Linda Gillberg, Vasiliki Skara, Eirini Antoniou, Erik Södersten, Per Svenningsson, Karima Chergui, Markus Ringnér, Thomas Perlmann, Johan Holmberg

This study shows that PRC2-mediated repression is essential for maintaining identity and function of dopaminergic and serotonergic neurons, with loss leading to behavioral deficits.

Science Advances · Aug 26, 2022Peer-reviewedNeuroscience

Activation of endogenous retroviruses during brain development causes neuroinflammation

Marie E. Jönsson, Raquel Garza, Yogita Sharma, Rebecca Petri, Erik Södersten, Jenny G Johansson, Pia Annette Johansson, Diahann A. M. Atacho, Karolina Pircs, Sofia Madsen, David Yudovich, Ramprasad Ramakrishnan, Johan Holmberg, Jonas Larsson, Patric Jern, Johan Jakobsson

This study shows that activation of endogenous retroviruses during brain development leads to neuroinflammation in adult mice.

bioRxiv · Jul 7, 2020 · PreprintNeuroscience