Author

Danny MacKenzie

New York Institute of TechnologyORCID 0000-0002-3632-6040Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:chromatin & epigenetic regulation· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1

Frequent Co-Authors

Alexis M. Perez· 1 shared Carl L. Schildkraut· 1 shared Dong ZhangiD· 1 shared Heba Zuhair AbidiD· 1 shared Kaitlin RaseleyiD· 1 shared Katherine Loomba· 1 shared Lars Udo-Bellner· 1 shared Manrose SinghiD· 1 shared

Activity by Year

2024-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

Alexis M. Perez

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1 paper

Recent·Most cited

Elucidation of the molecular mechanism of the breakage-fusion-bridge (BFB) cycle using a CRISPR-dCas9 cellular model

Manrose Singh, Kaitlin Raseley, Alexis M. Perez, Danny MacKenzie, Settapong T Kosiyatrakul, Sanket Desai, Noelle Batista, Navjot Guru, Katherine Loomba, Heba Zuhair Abid, Yilin Wang, Lars Udo-Bellner, Randy F. Stout, Carl L. Schildkraut, Ming Xiao, Dong Zhang

This study uses a CRISPR-dCas9 model to investigate the BFB cycle, revealing increased DNA damage, chromosome fusions, and involvement of DNA repair pathways in ITCB formation.

Nucleic Acids Research · Aug 28, 2024Peer-reviewedCancer Biology