Author

B.S. Aswathy

Stony Brook UniversityORCID 0000-0002-3763-3448Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Benjamin KolisnykiD· 1 shared Emily J. ParkiD· 1 shared Jacqueline MartiniD· 1 shared Jason Ni· 1 shared Jonathan Plessis‐BelairiD· 1 shared Jordan A. PearsoniD· 1 shared Lorenz StuderiD· 1 shared Markus RießlandiD· 1 shared

Activity by Year

2024-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Benjamin KolisnykORCID

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1 paper

Recent·Most cited

The SATB1-MIR22-GBA axis mediates glucocerebroside accumulation inducing a cellular senescence-like phenotype in dopaminergic neurons

Taylor Russo, Benjamin Kolisnyk, B.S. Aswathy, Tae Wan Kim, Jacqueline Martin, Jonathan Plessis‐Belair, Jason Ni, Jordan A. Pearson, Emily J. Park, Roger B. Sher, Lorenz Studer, Markus Rießland

This study identifies a gene regulatory pathway involving SATB1, MIR22HG, and GBA that leads to glucocerebroside accumulation and cellular senescence in dopaminergic neurons, contributing to Parkinson's Disease and aging.

Aging Cell · Feb 1, 2024Peer-reviewedNeuroscience