Author

Taylor Russo

Stony Brook UniversityORCID 0000-0003-4564-5366Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Assay:bulk RNA-seq· 2 Research Focus:gene regulatory network inference· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

Markus RießlandiD· 4 shared Jonathan Plessis‐BelairiD· 3 shared Roger B. SheriD· 3 shared B.S. AswathyiD· 1 shared Benjamin KolisnykiD· 1 shared Emily J. ParkiD· 1 shared Jacqueline MartiniD· 1 shared Jason Ni· 1 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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4 shared papersNeuroscience

Markus RießlandORCID

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4 papers

Recent·Most cited

Characterizing the SASP-Dependent Paracrine Spreading of Senescence Between Human Brain Cell Types

Taylor Russo, Markus Rießland

The paper identifies cell-type-specific SASP ligand-receptor pairs that mediate paracrine senescence spreading in human brain cells and shows that targeting these factors can prevent senescence spread.

Aging Cell · Aug 1, 2026Peer-reviewedNeuroscience

Nuclear Import Defects Drive Cell Cycle Dysregulation in Neurodegeneration

Jonathan Plessis‐Belair, Taylor Russo, Markus Rießland, Roger B. Sher

This study shows that nuclear import defects drive cell cycle dysregulation in neurodegeneration by impairing nuclear import pathways and causing cell cycle re-entry in neurons.

Aging Cell · May 16, 2025Peer-reviewedNeuroscience

Regulatory Network Inference of Induced Senescent Midbrain Cell Types Reveals Cell Type-Specific Senescence-Associated Transcriptional Regulators

Taylor Russo, Jonathan Plessis‐Belair, Roger B. Sher, Markus Rießland

The paper identifies cell type-specific senescence-associated transcriptional regulators in midbrain cell lines using PCA and regulatory network inference.

bioRxiv · Feb 6, 2025 · PreprintNeuroscience

The SATB1-MIR22-GBA axis mediates glucocerebroside accumulation inducing a cellular senescence-like phenotype in dopaminergic neurons

Taylor Russo, Benjamin Kolisnyk, B.S. Aswathy, Tae Wan Kim, Jacqueline Martin, Jonathan Plessis‐Belair, Jason Ni, Jordan A. Pearson, Emily J. Park, Roger B. Sher, Lorenz Studer, Markus Rießland

This study identifies a gene regulatory pathway involving SATB1, MIR22HG, and GBA that leads to glucocerebroside accumulation and cellular senescence in dopaminergic neurons, contributing to Parkinson's Disease and aging.

Aging Cell · Feb 1, 2024Peer-reviewedNeuroscience