Author

Amrita A. Iyer

Baylor College of MedicineORCID 0000-0002-3860-6863Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:ATAC-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Andrew K. GrovesiD· 1 shared Hongyuan ZhangiD· 1 shared Hsin‐I Jen· 1 shared Ishwar HosamaniiD· 1 shared Jenny Jia SuniD· 1 shared John D Nguyen· 1 shared Lisa A. BeyeriD· 1 shared Melissa M. McGoverniD· 1 shared

Activity by Year

2022-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperDevelopmental Biology

Andrew K. GrovesORCID

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1 paper

Recent·Most cited

Cellular reprogramming with ATOH1, GFI1, and POU4F3 implicate epigenetic changes and cell-cell signaling as obstacles to hair cell regeneration in mature mammals

Amrita A. Iyer, Ishwar Hosamani, John D Nguyen, Tiantian Cai, Sunita Singh, Melissa M. McGovern, Lisa A. Beyer, Hongyuan Zhang, Hsin‐I Jen, Rizwan Yousaf, Onur Birol, Jenny Jia Sun, Russell Scott Ray, Yehoash Raphael, Neil Segil, Andrew K. Groves

This study shows that reprogramming mature mammalian cochlear cells with ATOH1, GFI1, and POU4F3 is hindered by epigenetic inaccessibility and cell-cell signaling, particularly Notch signaling.

eLife · Nov 17, 2022Peer-reviewedDevelopmental Biology