Author

Gunnar Goerges

University Hospital MünsterORCID 0000-0002-3952-320XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Pharmacology & Toxicology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Assay:electrophysiology· 1 Organism:human· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Guiscard SeebohmiD· 2 shared Julian Alexander SchreiberiD· 2 shared Aladdin ElgendyiD· 1 shared André P. ZeipeltiD· 1 shared Andrea Ulrike SteinbickeriD· 1 shared Arie O. VerkerkiD· 1 shared Daniel TapkeniD· 1 shared Daniel TödtiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersPharmacology & Toxicology

Guiscard SeebohmORCID

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2 papers

Recent·Most cited

A sequence variation between two orthologues alters functional expression of the potassium channel Kesv of Ectocarpus siliculosus virus

Purva Asrani, Aladdin Elgendy, André P. Zeipelt, Gunnar Goerges, Julian Alexander Schreiber, Richard J. P. Brown, Daniel Tödt, Daniel Tapken, Lars Schaefer, Guiscard Seebohm, Raphael Stoll

This study compares the functional expression of two potassium channels, Kesv 1 and Kesv 2, identifying sequence differences that affect ion conductance and drug sensitivity.

bioRxiv · Aug 5, 2026 · PreprintPharmacology & Toxicology

TMPRSS6 cleaves KCNE1 and causes arrhythmias in iron overload disease

Stefan Peischard, Philipp Kastl, Gunnar Goerges, Julian Alexander Schreiber, Arie O. Verkerk, Ronald Wilders, Paul Disse, Isabelle Hornung, Ursula Klingmüller, Andrea Ulrike Steinbicker, Martina Rauner, Maja Vujić Spasić, Frank Rosenbauer, Sven Guenther Meuth, Thomas Budde, , Guiscard Seebohm

This study identifies TMPRSS6 cleavage of KCNE1 as a mechanism causing arrhythmias in iron overload disease using human iPSC-derived cardiomyocytes and computational models.

bioRxiv · Sep 4, 2024 · PreprintPhysiology