Author

Yael Almog

Tel Aviv UniversityORCID 0000-0002-4290-9779Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Assay:electronic health records· 2 Organism:mouse· 2 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

Marina Brusel· 2 shared Moran RubinsteiniD· 2 shared Anat MavashoviD· 1 shared Igor UlitskyiD· 1 shared Karen Anderson· 1 shared Karni Vilian· 1 shared Rotem Ben‐Tov PerryiD· 1 shared Shaked Turk· 1 shared

Activity by Year

2019-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersNeuroscience

Marina Brusel

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Alterations in background ECoG activity and behavioral deficits in a mouse model of CHD2-related developmental delay

Anat Mavashov, Shaked Turk, Yael Sarusi, Marina Brusel, Rotem Ben‐Tov Perry, Shir Quinn, Yael Almog, Karni Vilian, Igor Ulitsky, Moran Rubinstein

This paper characterizes behavioral and epileptic phenotypes in a CHD2 mutant mouse model, identifying genetic modifiers and altered ECoG activity associated with developmental delay.

bioRxiv · Mar 18, 2025 · PreprintNeuroscience

Early hippocampal hyperexcitability followed by disinhibition in a mouse model of Dravet syndrome

Yael Almog, Marina Brusel, Karen Anderson, Moran Rubinstein

The paper analyzes the activity of hippocampal neurons in a mouse model of Dravet syndrome across different disease stages, finding temporary excitability increases in pyramidal neurons and persistent inhibition deficits in interneurons.

bioRxiv · Oct 3, 2019 · PreprintNeuroscience