Alexey V. Revtovich

This study shows that P. aeruginosa liquid-based pathogenesis induces metabolic changes in C. elegans, involving lipid droplet depletion and HLH-30-dependent rewiring.

The paper identifies small molecules targeting AML by screening for compounds with predicted functions in apoptosis, glutathione metabolism, and autophagy, validating their efficacy in AML cells and patient-derived samples.

This study identifies five compounds that enhance Caenorhabditis elegans resistance to multiple pathogens by activating host defense pathways.

This study shows that a dietary vitamin B12 deficiency in C. elegans impairs mitochondrial function and stress resistance, which can be reversed by B12 supplementation.