Author

Adam Briner

The University of QueenslandORCID 0000-0002-4760-5037Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Approach:biophysical modeling· 1 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

Aaron D. GitleriD· 2 shared Garam KımiD· 2 shared Alex S. HolehouseiD· 1 shared Avinash Fernandes· 1 shared Beryl B. CummingsiD· 1 shared Björn OskarssoniD· 1 shared Borna NovakiD· 1 shared Bradley F. BoeveiD· 1 shared

Activity by Year

2022-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

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2 shared papersCell Biology

Aaron D. GitlerORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Cell BiologybioRxivPreprintAug 24, 2026

Lipoproteins modulate the uptake and biological functionof cationic cell-penetrating peptides across the animal lineage.

Sovanny R. Taylor, Temilade V. Oluwasesin, Muthu Raj Salaikumaran…·Steven Boeynaems

This study identifies lipoprotein-mediated endocytosis as a conserved mechanism for cationic cell-penetrating peptides across animal lineages, showing it modulates their antimicrobial function.

DOI 10.64898/2026.08.23.746575

humanmouse

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NeuroscienceLife Science AlliancePeer-reviewedOct 31, 2022

Genome-wide CRISPRi screening reveals regulators of Alzheimee's tau pathology shared between exosomal and vesicle-free tau seeds.

Juan Carlos Polanco, Yevhen A. Akimov, Avinash Fernandes…·Jürgen Götz

This study uses CRISPRi screening to identify cellular regulators that restrict both exosomal and vesicle-free tau seeding in Alzheimer's disease.

12 citations

humanCRISPR screen

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NeuroscienceNaturePeer-reviewedFeb 23, 2022

TDP-43 represses cryptic exon inclusion in FTD/ALS gene UNC13A

X. Rosa Ma, Mercedes Prudencio, Yuka Mitsuhashi Koike…·Aaron D. Gitler

This study shows that TDP-43 represses a cryptic exon in UNC13A, and genetic variants associated with FTD/ALS increase cryptic exon inclusion when TDP-43 is dysfunctional.

574 citationstop 1%

humanbulk RNA-seq

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