Author

Maria M. Usowicz

University of BristolORCID 0000-0002-4844-4546Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:Drosophila· 2 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

James J. L. HodgeiD· 2 shared Simon A. LoweiD· 2 shared Carlo NG GiachelloiD· 1 shared Dimitri Michael KullmanniD· 1 shared Henry HouldeniD· 1 shared James E. RothmaniD· 1 shared James E.C. JepsoniD· 1 shared N PateliD· 1 shared

Activity by Year

2017-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersNeuroscience

James J. L. HodgeORCID

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2 papers

Recent·Most cited

Neuronal overexpression of DYRK1A/minibrain alters motor decline, neurodegeneration and synaptic plasticity in Drosophila

Simon A. Lowe, Maria M. Usowicz, James J. L. Hodge

This study shows that overexpressing the Drosophila DYRK1A ortholog, minibrain, in neurons leads to motor decline, neurodegeneration, and synaptic changes similar to Down syndrome features.

Neurobiology of Disease · Jan 29, 2019Peer-reviewedNeuroscience

Mutations In Membrin/GOSR2 Reveal Stringent Secretory Pathway Demands Of Dendritic Growth And Synaptic Integrity

Roman Praschberger, Simon A. Lowe, Nancy T. Malintan, Carlo NG Giachello, N Patel, Henry Houlden, Dimitri Michael Kullmann, Richard Baines, Maria M. Usowicz, Shyam Sundar Krishnakumar, James J. L. Hodge, James E. Rothman, James E.C. Jepson

This study investigates how mutations in Membrin/GOSR2 affect dendritic growth and synaptic integrity in Drosophila models, revealing their role in neurological disorders.

Cell Reports · Oct 1, 2017Peer-reviewedNeuroscience