Author

Carlos Ramón Morales

McGill UniversityORCID 0000-0002-4882-870XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:imaging· 1 Assay:proteomics· 1

Frequent Co-Authors

Alexey V. PshezhetskyiD· 3 shared Éric BonneiliD· 2 shared Erjun ZhangiD· 2 shared Graziella Di CristoiD· 2 shared Gregory Anton LodygenskyiD· 2 shared Mahsa TaherzadehiD· 2 shared Pierre ThibaultiD· 2 shared Poulomee BoseiD· 2 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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3 shared papersNeuroscience

Alexey V. PshezhetskyORCID

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3 papers

Recent·Most cited

GeneticsbioRxivPreprintFeb 24, 2026

Synthetic analogue of adrenocorticotropic hormone, ACTH(4-7)PGP delays neurological manifestations in diseases of mucopolysaccharidosis III spectrum by reducing neuroinflammation and rescuing neurotransmission, synaptogenesis, and axonal demyelination

Travis Mooree, Patricia Dubot, Gustavo Monteiro Viana…·Alexey V. Pshezhetsky

The paper tests a synthetic peptide ACTH(4-7)PGP in MPS III mouse models, showing it improves synaptic function, reduces neuroinflammation, and extends lifespan.

DOI 10.64898/2026.02.20.707013

mouseexperimental assay

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NeuroscienceFrontiers in Molecular NeurosciencePeer-reviewedDec 12, 2023

Severe Central Nervous System Demyelination in Sanfilippo Disease

Mahsa Taherzadeh, Erjun Zhang, Irene Londoño…·Alexey V. Pshezhetsky

This study investigates white matter injury in Sanfilippo disease using mouse models and human samples, revealing demyelination and specific MRI markers for disease progression.

10 citations

mousehuman

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NeurosciencebioRxivPreprintJul 6, 2020

Early defects in lysosomal storage diseases disrupt excitatory synaptic transmission

Camila De Britto Pará De Aragão, Luigi Bruno, Poulomee Bose…·Alexey V. Pshezhetsky

This study shows that lysosomal storage diseases cause synaptic structure and neurotransmission defects before cognitive symptoms appear in mice and humans.

DOI 10.1101/2020.07.06.186809

mousehuman

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