Author

Anna K. Golebiewski

University of ArizonaORCID 0000-0002-4952-4812Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Research Focus:differential expression· 3 Organism:human· 3 Approach:statistical model· 3 Assay:ATAC-seq· 2 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Approach:experimental assay· 1

Frequent Co-Authors

Casey E. RomanoskiiD· 3 shared Lindsey K. StolzeiD· 3 shared Aarthi RavindraniD· 1 shared Alhan Mehrabi Yazdi· 1 shared Anu ToropaineniD· 1 shared Austin C. ConkliniD· 1 shared Binh T. Tran· 1 shared Cecilia M. Careaga· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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3 shared papersGenetics

Casey E. RomanoskiORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Inflammation-Induced Alternative Splicing in Human Endothelial Cells Reveals Genetic Mechanisms of Cardiovascular Disease Risk

Anna K. Golebiewski, Lindsey K. Stolze, Valentina D. Vazquez, Alhan Mehrabi Yazdi, Cecilia M. Careaga, Casey E. Romanoski

This study identifies inflammation-induced alternative splicing in human endothelial cells and links it to cardiovascular disease risk through genetic mechanisms.

bioRxiv · Jul 29, 2025 · PreprintGenetics

Coronary Artery Disease risk variant dampens the expression of CALCRL by reducing HSF binding to shear stress responsive enhancer in endothelial cells

Ilakya Selvarajan, Miika Kiema, Ru‐Ting Huang, Jin Li, Jiayu Zhu, Petri Pölönen, Tiit Örd, Kadri Õunap, Mehvash Godiwala, Anna K. Golebiewski, Aarthi Ravindran, Kiira Mäklin, Anu Toropainen, Lindsey K. Stolze, Maximiliano Arce, , Minna Unelma Kaikkonen

The paper identifies a CAD risk SNP rs880890 in an endothelial enhancer that reduces CALCRL expression by impairing HSF binding under shear stress.

Arteriosclerosis Thrombosis and Vascular Biology · Apr 11, 2024Peer-reviewedGenetics

Single cell omic profiles of human aortic endothelial cells in vitro and human atherosclerotic lesions ex vivo reveals heterogeneity of endothelial subtype and response to activating perturbations

Maria L. Adelus, Jiacheng Ding, Binh T. Tran, Austin C. Conklin, Anna K. Golebiewski, Lindsey K. Stolze, Michael Bernard Whalen, Darren A. Cusanovich, Casey E. Romanoski

This study profiles human aortic endothelial cells in vitro and atherosclerotic lesions ex vivo, revealing heterogeneous subtypes and their responses to perturbations.

bioRxiv · Apr 3, 2023 · PreprintGenomics