Author
Nitin Mohan
Indian Institute of Technology KanpurORCID 0000-0002-5168-1462Unclaimed ORCID profile · follow to get notified of new preprints
Biology Disciplines
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2024-2026 · peak 3/yrCo-Author Collaboration Network
10 top collaborators · 11 mutual co-author links6 papers
Recent·Most cited
- NeuroscienceAlzheimer s Research & TherapyPeer-reviewed
Stress-Induced Alteration of Small Extracellular Vesicles Drives Amyloid-Beta Sequestration and Exacerbates Alzheimer's Disease Pathogenesis
Sadaqa Zehra, Sanskriti Rai, Komal Rani…·Saroj Kumar
This study shows stress-induced changes in small extracellular vesicles (sEVs) enhance their binding to amyloid-beta aggregates, promoting plaque formation and disease progression in Alzheimer's.
- Cell BiologybioRxivPreprint
MAP4-MAP7D1 partitioning on tyrosinated-detyrosinated microtubules coordinates lysosome positioning in nutrient signalling
Deepak M Khushalani, Joydipta Kar, Satya Bikash Nayak…·Nitin Mohan
This study shows how MAP4 and MAP7D1 partition on different microtubule subsets to regulate lysosome positioning in response to nutrient availability.
1 citation - Cell BiologybioRxivPreprint
mTORC1 signaling modulate microtubule tyrosination/detyrosination status to regulate lysosome dynamics
Jeganath Ammavasai, Nitin Mohan
This study shows mTORC1 signaling regulates lysosome dynamics by modulating microtubule tyrosination/detyrosination, impacting cancer cell migration and EMT.
- Cell BiologybioRxivPreprint
Suppressing microtubule detyrosination augments AAV2 endosomal escape and gene delivery.
Shefali Tripathi, Shamshul Huda, Joydipta Kar…·Nitin Mohan
This study shows that inhibiting microtubule detyrosination improves AAV2 endosomal escape and gene delivery in cells and mouse models.
DOI 10.1101/2025.08.12.669862 - Developmental BiologybioRxivPreprint
Paradoxical Tumor Suppressor Role of Yorkie Through a TOR-Dependent α-Tubulin Acetylation in Select Squamous Epithelium
Rachita Bhattacharya, Jeganath Ammavasai, Shruti Agarwal…·Nitin Mohan
The paper shows that Yorkie acts as a tumor suppressor in Drosophila squamous epithelia by suppressing TOR-driven α-tubulin acetylation, which is tissue-specific and distinct from other epithelial lineages.
1 citation