Author

Galit Lahav

Harvard UniversityORCID 0000-0002-5473-3686Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:imaging· 1

Frequent Co-Authors

Ashwini JambhekariD· 2 shared Bas PonsioeniD· 1 shared Chengzhe TianiD· 1 shared Clemens B. HugiD· 1 shared Fabian FröhlichiD· 1 shared Hugo J.G. SnippertiD· 1 shared José Santos ReyesiD· 1 shared Kotaro FujimakiiD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersCell Biology

Ashwini JambhekarORCID

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2 papers

Recent·Most cited

Cell BiologyCell ReportsPeer-reviewedSep 27, 2025

DNA damage checkpoints balance a tradeoff between diploid- and polyploid-derived arrest failures

Kotaro Fujimaki, Ashwini Jambhekar, Galit Lahav

This study shows DNA damage checkpoints balance a tradeoff between reducing arrest failure in diploid and polyploid cells, with optimal checkpoint strengths being suboptimal for each failure type.

4 citations

humanimaging

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Systems BiologybioRxivPreprintOct 6, 2022

Multi range ERK responses shape the proliferative trajectory of single cells following oncogene induced senescence

Jia-Yun Chen, Clemens B. Hug, José Santos Reyes…·Galit Lahav

The paper investigates how varying ERK activity levels influence proliferation in cells undergoing oncogene-induced senescence, finding intermediate ERK activity promotes maximal proliferation.

1 citation

humanbulk RNA-seq

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