Author

Seyyed Hani Moussavi Nik

The University of AdelaideORCID 0000-0002-5727-6863Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Bioinformatics· 1 Developmental Biology· 1 Genomics· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Approach:experimental assay· 3 Organism:zebrafish· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 1 Research Focus:protein structure prediction· 1

Frequent Co-Authors

Michael T. LardelliiD· 3 shared Morgan NewmaniD· 3 shared Amanda L. LumsdeniD· 2 shared Nhi HiniD· 2 shared Alastair J. LudingtoniD· 1 shared Alon M. DouekiD· 1 shared Angela S. LairdiD· 1 shared Caitlin W. Lucas· 1 shared

Activity by Year

2018-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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3 shared papersGenomics

Michael T. LardelliORCID

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3 papers

Recent·Most cited

GenomicsHuman Molecular GeneticsPeer-reviewedJun 16, 2020

Accelerated loss of hypoxia response in zebrafish with familial Alzheimer's disease-like mutation of Presenilin 1.

Morgan Newman, Seyyed Hani Moussavi Nik, Greg Trevor Sutherland…·Michael T. Lardelli

This study shows that zebrafish with PSEN1 mutations exhibit accelerated hypoxia response loss with age, suggesting a link between PSEN1 dysfunction and Alzheimer's disease pathogenesis.

15 citations

zebrafishhuman

BioinformaticsPLoS ONEPeer-reviewedJan 24, 2020

Accelerated brain aging towards transcriptional inversion in a zebrafish model of familial Alzheimer’s disease

Nhi Hin, Morgan Newman, Jan Kaslin…·Michael T. Lardelli

This study uses a zebrafish model of familial Alzheimer’s disease to show accelerated brain aging and transcriptional inversion linked to glucocorticoid resistance and immune pathway changes before histopathology.

42 citationstop 5%

zebrafishbulk RNA-seq

Developmental BiologybioRxivPreprintJan 2, 2018

HMGA1 zebrafish co-orthologue hmga1b can modulate p53-dependent cellular responses but is unable to control the alternative splicing of psen1

Seyyed Hani Moussavi Nik, Morgan Newman, Amanda L. Lumsden…·Michael T. Lardelli

The paper shows that hmga1b in zebrafish can modulate p53-dependent cellular responses but not alternative splicing of psen1, similar to hmga1a.

DOI 10.1101/241927

zebrafishexperimental assay