Lakeisha A. Lewter

This study identifies CGRPR-expressing neurons in the central amygdala as key players in pain hypersensitivity, showing their role in both injury-induced and spontaneous pain responses.

This study investigates the role of amygdala CGRP receptors in persistent bladder pain in mice, showing that right CeA inhibition reduces pain-like behavior while left inhibition sustains hypersensitivity.

This study investigates the role of CeA CGRP in neuropathic pain, finding that it modulates cold sensitivity differently based on injury type and hemisphere.

The paper identifies a parabrachial-to-amygdala circuit mediated by CGRP that drives hemispheric lateralization of visceral pain processing in mice.