Yong‐Seok Lee

Growth hormone is required for hippocampal engram cell maturation

This study identifies growth hormone as essential for hippocampal engram cell maturation by facilitating synaptic strengthening during memory formation.

Hippocampal-cortical interactions in the consolidation of social memory

This study identifies specific hippocampal-cortical circuits involved in social memory consolidation using optogenetic and imaging techniques.

Cerebellar systems consolidation driven by the temporal dynamics of Purkinje cell excitability

The paper investigates the role of Purkinje cell excitability in systems consolidation, finding that manipulating excitability within a specific time window after learning affects memory retention.

Acquisition phase-specific contribution of climbing fiber transmission to cerebellum-dependent motor memory

This study investigates the role of climbing fiber transmission in different phases of motor memory formation and finds that it is critical for memory acquisition but not for consolidation or retrieval.

Cerebellar Bergmann Glia Integrating Noxious Information Modulate Nocifensive Behaviors

The paper identifies Bergmann glia in the cerebellum as mediators of noxious information processing and their role in modulating pain-related behaviors.

Social isolation impairs the prefrontal- nucleus accumbens circuit subserving social recognition in mice

This study shows that early social isolation in mice impairs a specific prefrontal-accumbens circuit involved in social recognition, and that activating this circuit can reverse the deficit.

Ablation of STAT3 in Purkinje Cells Reorganizes Cerebellar Synaptic Plasticity in Long-Term Fear Memory Network

This study shows that deleting STAT3 in Purkinje cells alters synaptic plasticity and enhances long-term fear memory.

Kv1.2 contributes to pattern separation by regulating the hippocampal CA3 neuronal ensemble size

This study shows that Kv1.2 regulates hippocampal CA3 neuronal ensemble size and plasticity, contributing to pattern separation by preventing excessive synaptic changes.