Suk‐Ho Lee

The paper investigates short-term facilitation at excitatory synapses in the rat prefrontal cortex, identifying Ca2+- and Syt7-dependent mechanisms that contribute to temporal associative learning.

This study identifies a mechanism by which prelimbic corticopontine neurons gate extinction learning through mitochondria-dependent post-tetanic potentiation.

This study investigates Kv1 channel downregulation in LGI1 knock-out mice, showing it occurs in somatodendritic regions and is linked to increased neuronal bursting and changes in the Kv1 interactome.

The paper investigates how CA3 pyramidal cell excitability is regulated by mossy fiber and perforant pathway inputs, showing their role in sequence learning through bidirectional regulation of excitability and synaptic plasticity.

This study investigates how subthreshold depolarization enhances glutamate release via L-type Ca2+ channels and Ca2+-dependent signaling molecules.

The paper shows that burst firing in hippocampal granule cells is necessary for Hebbian LTP at lateral perforant path synapses, but not at medial perforant path synapses.

The paper investigates whether voltage-gated calcium channels (VGCCs) contribute to spontaneous glutamate release, finding that coupling distance and calmodulin influence this contribution in different synapses.

This study shows calbindin regulates Kv4.1 trafficking and neuronal excitability via CaMKII-dependent phosphorylation, linking Ca2+ buffering to neuronal function.

This study shows that Tg2576 mice exhibit impaired pattern separation linked to hyperexcitable dentate gyrus due to Kv4.1 downregulation, which is reversible with antioxidant treatment.

This study shows that Kv1.2 regulates hippocampal CA3 neuronal ensemble size and plasticity, contributing to pattern separation by preventing excessive synaptic changes.

This study identifies Kv4.1 as a key ion channel regulating low-frequency firing in mature dentate granule cells, essential for pattern separation.

This study shows that dendritic Na+ spikes are necessary for LTP at perforant path-granule cell synapses in rat hippocampal slices.