Author

José Manuel García-Martínez

Universidad Rey Juan CarlosORCID 0000-0002-6498-5174Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

Ana Chocarro‐CalvoiD· 2 shared Colin R. GodingiD· 2 shared Custodia García‐JiméneziD· 2 shared Javier Martínez‐UserosiD· 2 shared Pakavarin LouphrasitthipholiD· 2 shared Alice Bellini· 1 shared Ana Ramírez-SáncheziD· 1 shared Colin KennyiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersCancer Biology

Ana Chocarro‐CalvoORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Functional specialization of MITF, TFEB and TFE3 drives radically distinct adaptive gene expression programs in melanoma.

Diogo Dias, Erica Aparecida de Oliveira, Román Martí-Díaz, Sarah Andrews, Ana Chocarro‐Calvo, Alice Bellini, Laura Mosteo, Yurena Vivas, Jagat Singh Chauhan, Linxin Li, José Manuel García-Martínez, José Neptuno Rodríguez‐López, Silvya Stuchi Maria–Engler, Colin Kenny, Javier Martínez‐Useros, , Colin R. Goding

The paper identifies distinct gene expression programs driven by MITF, TFEB, and TFE3 in melanoma, revealing their functional specialization in regulating differentiation, metabolism, and immune infiltration.

Cell Reports · Nov 22, 2025Peer-reviewedCancer Biology

Metabolic buffering suppresses phenotype switching in cancer

Ana Ramírez-Sánchez, Miguel Jociles-Ortega, José Manuel García-Martínez, Irene Torrens-Martínez, Javier Martínez‐Useros, María Jesús Fernández‐Aceñero, Teresa Olmos-De Blas, Pakavarin Louphrasitthiphol, Colin R. Goding, Custodia García‐Jiménez, Ana Chocarro‐Calvo

This study shows that glycogen storage and metabolic buffering suppress phenotype switching in melanoma, with low glycogen levels linked to invasion and poor survival.

bioRxiv · Apr 7, 2025 · PreprintCancer Biology