Author

Chelsea Marie T. Parrocha

University of California, IrvineORCID 0000-0002-6502-1297Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Neuroscience· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:metabolomics· 1

Frequent Co-Authors

Adam G. KreutzeriD· 1 shared Alvin PhaniD· 1 shared Amal AlachkariD· 1 shared Angele De SilvaiD· 1 shared Charity Henrich· 1 shared Elizabeth HeadiD· 1 shared Emily Shapiro· 1 shared Geoffrey Winston AbbottiD· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperNeuroscience

Adam G. KreutzerORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Antibodies Raised Against an Aβ Oligomer Mimic Recognize Pathological Features in Alzheimer's Disease and Associated Amyloid-Disease Brain Tissue

Adam G. Kreutzer, Chelsea Marie T. Parrocha, Sepehr Haerianardakani, Gretchen Guaglianone, Jennifer T. Nguyen, Michelle N. Diab, William H. Yong, Mari A. Perez-Rosendahl, Elizabeth Head, James S. Nowick

The paper creates an Aβ trimer mimic and studies antibodies against it, showing they recognize pathological features in Alzheimer's disease brain tissue.

ACS Central Science · Dec 21, 2023Peer-reviewedNeuroscience

Intergenerational Stress Transmission is Associated with Brain Metabotranscriptome Remodeling andMitochondrial Dysfunction

Sammy Alhassen, Siwei Chen, Lamees Alhassen, Alvin Phan, Mohammad Khoudari, Angele De Silva, Huda Barhoosh, Zitong Wang, Chelsea Marie T. Parrocha, Emily Shapiro, Charity Henrich, Zicheng Wang, Léon Mutesa, Pierre Baldi, Geoffrey Winston Abbott, Amal Alachkar

This study shows that prenatal stress exposure in mice leads to behavioral deficits in offspring, associated with mitochondrial dysfunction and epigenetic changes, which can be mitigated by acetyl-L-carnitine.

Communications Biology · Jun 24, 2021Peer-reviewedNeuroscience