Author

Haley N. Janowitz

Pennsylvania State UniversityORCID 0000-0002-6508-7237Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Bioengineering· 1 Genetics· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:drug response / drug discovery· 2 Organism:human· 2 Organism:mouse· 2 Approach:statistical model· 2 Assay:ATAC-seq· 1 Organism:C. elegans· 1 Approach:deep learning· 1 Assay:electronic health records· 1

Frequent Co-Authors

Adam R. FentoniD· 1 shared Agnès TrébuchoniD· 1 shared Andrew C. MerceriD· 1 shared Angélique PeretiD· 1 shared Anne Fabienne LepineiD· 1 shared April R. Giles· 1 shared Bilge Esin ÖztürkiD· 1 shared Brett C. Hogle· 1 shared

Activity by Year

2017-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperGenetics

Adam R. FentonORCID

Author profile →

3 papers

Recent·Most cited

BioengineeringbioRxivPreprintMay 17, 2026

Structural dynamics insights into principles underlying the fitness of new broadly potent AAVs

Molly E. Johnson, Bilge Esin Öztürk, Thomas H. Tugwell…·Leah C. Byrne

The paper develops a new AAV engineering workflow, scAAVengr-Hunt, to create potent vectors and identifies a structural mechanism underlying their broad species and tissue potency.

1 citation

non-human primatemouse

𝕏 PostBluesky

NeuroscienceAnnals of NeurologyPeer-reviewedJun 21, 2023

GluK2 is a target for gene therapy in drug-resistant Temporal Lobe Epilepsy

Céline Boileau, Séverine Deforges, Angélique Peret…·Valérie Crépel

This study demonstrates that gene therapy targeting GluK2 reduces seizures in TLE models and patient-derived slices.

33 citationstop 2%

𝕏 PostBluesky

GeneticsbioRxivPreprintAug 29, 2017

A Caenorhabditis elegans model of adenylosuccinate lyase deficiency reveals neuromuscular and reproductive phenotypes of distinct etiology

Adam R. Fenton, Haley N. Janowitz, Melanie R. McReynolds…·Wendy Hanna‐Rose

This study uses a C. elegans model to show that adenylosuccinate lyase deficiency causes distinct neuromuscular and reproductive defects with different metabolic causes.

4 citations

C. elegansstatistical model

𝕏 PostBluesky