Vonetta L. Edwards

This study shows that female sex hormones enhance gonococcal colonization at the endocervix by altering mucus production and structure, increasing bacterial microcolony size and number.

This paper develops a microphysiologic model of the cervical epithelium that replicates microbial, immunologic, and pathogenic features of STIs caused by Chlamydia and Neisseria.

This study uses dual-species RNA-seq to analyze the transcriptional responses of Neisseria gonorrhoeae and human neutrophils during infection, revealing shared metabolic and immunologic processes.