David A. A. Baranger

This paper identifies childhood phenotypes associated with genetic risk for elevated CRP using a PheWAS in the ABCD Study, finding associations with weight, eating, and screen time that are largely independent of BMI.

This study performs a multi-ancestral GWAS of DSM-defined nicotine dependence, finding genetic correlations with other substance use disorders and health traits, and identifying a strong general addiction liability factor.

This study investigates genetic correlations and causal relationships between schizophrenia, cannabis use disorder, and tobacco smoking across African and European ancestries, identifying pleiotropic loci and convergent genetic effects.

The paper identifies a GRPR-expressing spinal neuron population that encodes itch and shows that kappa opioid receptor agonists inhibit itch by targeting these neurons.

The paper shows that multi-omics integration fails to find true-positive novel genes from underpowered GWAS of brain-related traits, highlighting the need for larger sample sizes.

This study examines the stability of fMRI reward-related activation over 1-2 years in adolescents and young adults, finding modest stability and suggesting that certain contrasts may be less effective for individual difference studies.

The paper reports a crowdsourced challenge to predict height from genome-wide genotyping data using the openSNP dataset, with the winner achieving an r2 of 0.53.

The paper identifies smaller brain volumes in the right dorsolateral prefrontal cortex and insula as predispositional risk factors for alcohol use, supported by genetic and longitudinal evidence.