Author

Renato Chaves Souto Branco

Indiana Biosciences Research InstituteORCID 0000-0002-6886-7693Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:mouse· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Addison S. Miller· 1 shared Andrew T. TempliniD· 1 shared Arianna Harris-KawanoiD· 1 shared Carmella Evans‐MolinaiD· 1 shared Chih‐Chun LeeiD· 1 shared Christine A. BerryhilliD· 1 shared Christopher ContrerasiD· 1 shared Christopher M. DavisiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperCell Biology

Addison S. Miller

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2 papers

Recent·Most cited

Cell BiologyCell Death and DiseasePeer-reviewedFeb 12, 2026

RIPK1 regulates beta-cell fate via actions on gene expression and kinase signaling in a mouse model of beta-cell self-reactivity

Christopher Contreras, Noyonika Mukherjee, Arianna Harris-Kawano…·Andrew T. Templin

This study shows RIPK1 promotes beta-cell death in autoimmune diabetes by affecting gene expression and kinase signaling, suggesting it as a therapeutic target.

DOI 10.1038/s41419-026-08471-0

mousebulk RNA-seq

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Molecular BiologyJournal of Biological ChemistryPeer-reviewedSep 27, 2024

miR-146a-5p mediates inflammation-induced β cell mitochondrial dysfunction and apoptosis

Preethi Krishnan, Renato Chaves Souto Branco, Staci A. Weaver…·Carmella Evans‐Molina

This study shows that miR-146a-5p overexpression in beta cells under inflammatory stress causes mitochondrial dysfunction and apoptosis, while its inhibition reverses these effects.

20 citationstop 1%

mousestatistical model

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