Author

Arianna Harris-Kawano

Indiana University School of MedicineORCID 0000-0002-5810-9274Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Addison S. Miller· 1 shared Andre De Oliveira· 1 shared Andrew T. TempliniD· 1 shared Christine A. BerryhilliD· 1 shared Christopher ContrerasiD· 1 shared Christopher M. DavisiD· 1 shared Debjyoti KunduiD· 1 shared Egan G. Mather· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperCell Biology

Addison S. Miller

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2 papers

Recent·Most cited

RIPK1 regulates beta-cell fate via actions on gene expression and kinase signaling in a mouse model of beta-cell self-reactivity

Christopher Contreras, Noyonika Mukherjee, Arianna Harris-Kawano, Egan G. Mather, Nansalmaa Amarsaikhan, Christopher M. Davis, Christine A. Berryhill, Madeline Peyton, Debjyoti Kundu, Kaitlyn A. Colglazier, Addison S. Miller, Renato Chaves Souto Branco, Travis S. Johnson, Steven P. Angus, Sylvaine You, , Andrew T. Templin

This study shows RIPK1 promotes beta-cell death in autoimmune diabetes by affecting gene expression and kinase signaling, suggesting it as a therapeutic target.

Cell Death and Disease · Feb 12, 2026Peer-reviewedCell Biology

Proinflammatory stress activates neutral sphingomyelinase 2 based generation of a ceramide-enriched β cell EV subpopulation

Zhiwei Xu, Irene Amalaraj, Andre De Oliveira, Arianna Harris-Kawano, Jacob R. Enriquez, Raghavendra G. Mirmira, Josie Eder, Meagan Burnet, Ivo Díaz Ludovico, Javier E. Flores, Ernesto Satoshi Nakayasu, Emily Kristen Sims

This study shows that proinflammatory stress in beta cells increases ceramide-enriched extracellular vesicles via nSMase2, which may affect paracrine signaling in diabetes.

Diabetes · Sep 2, 2025Peer-reviewedMolecular Biology